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Researchers with the UC Irvine-led study sample dust at the southern edge of the Salton Sea. Joe C. Wen School of Population & Public Health Photo Credit: Courtesy of University of California, Irvine |
Children living near the Salton Sea, in Southern California’s desert region of Imperial County, are experiencing poorer lung function than children exposed to less wind-blown dust, according to a new study led by researchers at the University of California, Irvine’s Joe C. Wen School of Population & Public Health.
They found that higher dust exposure – measured in hours per year – was linked to lower lung function, with the negative effects most pronounced among children living closest to the lake. The work, published in the American Journal of Respiratory and Critical Care Medicine, marks one of the first investigations to directly link dust events from a drying saline lake to measurable declines in children’s respiratory health.
A federal grant from the National Institute of Environmental Health Sciences and the Southern California Environmental Health Sciences Center funded the research in partnership with the Imperial Valley community-based organization Comite Civico del Valle.
From 2019 to 2022, investigators used spirometry on nearly 500 children, all around 10 years of age, to measure lung function. The test evaluates lung size and strength by gauging how much air a person can exhale, as well as how fast they can do so.
Team members collected nearly 1,300 lung function assessments, alongside health questionnaires and in-person clinical examinations. Using data from 12 air monitors maintained by the California Air Resources Board, they estimated participants’ exposure to particulate matter during dust events – defined as hours in which particulate matter concentrations exceeded regulatory thresholds. For each child, the researchers calculated cumulative dust event exposure during the three months preceding each lung function test.
The analysis revealed a clear association: Children living near the Salton Sea experienced worse lung function due to their exposure to dust events. The study builds on growing evidence that the high particulate matter levels around the shrinking Salton Sea contribute to elevated rates of asthma, wheezing and other respiratory conditions – echoing health disasters such as “Dust Bowl pneumonia” in the 1930s, when widespread dust exposure led to severe and often fatal respiratory illness.
“The drying of the Salton Sea is not only an environmental crisis but also a public health crisis,” said corresponding author Jill Johnston, associate professor of environmental and occupational health at Wen Public Health. “Our study provides concrete evidence that children in surrounding communities are facing measurable harm to their lungs as a result of increased dust exposure.”
California’s largest inland lake, the Salton Sea has been receding for decades, exposing large stretches of dried lakebed that release dust into the air when disturbed by wind. This dust can carry contaminants including pesticides, metals and other toxic substances. Communities near the lake, predominantly low-income and Latino, are disproportionately bearing the health burden.
In the article, the researchers emphasize the urgent need for continued monitoring and intervention to mitigate air pollution in the region. “Protecting the health of children in the Salton Sea communities requires immediate attention through targeted public health strategies,” Johnston said.
The study adds critical evidence to the body of research on environmental health risks linked to climate change and ecosystem decline. As similar changes in inland lakes occur globally, these potential impacts demand greater public health attention, underscoring the importance of preventive policies and community-level protections.
Title: Dust Events and Children’s Lung Function Near a Drying Saline Lake
Authors: Fangqi Guo, Elizabeth M Kamai, Sandrah P Eckel, Luis Olmedo, Esther Bejarano, Christian Torres, Christopher Zuidema, Edmund Seto, Shohreh F. Farzan, and Jill E. Johnston
Source/Credit: University of California, Irvine
Reference Number: med101425_01